Anti-leucine rich glioma inactivated 1 protein and anti-N-methyl-D-aspartate receptor encephalitis show distinct patterns of brain glucose metabolism in 18F-fluoro-2-deoxy-d-glucose positron emission tomography

Table 4 ¹⁸ F-fluoro-2-deoxy-d-glucose positron emission tomography in patients with anti-leucine rich glioma inactivated 1 protein encephalitis

Hypermetabolism	Hypometabolism
Cerebellum, bilat.	Cingulum ant. bilat., mid. frontal sup. med., med. orbital right
(2286 voxels)	(232 voxels in BA 10, 11, 24, 32)
Putamen, pallidum right
(322 voxels)
Precentral, frontal mid., sup. right
(210 voxels in BA 6, 8)
Putamen, pallidum left
(191 voxels)
Occipital inf., mid., calcarine right
(191 voxels in BA 17, 18, 19)
Parietal supramarginal, angular, temporal sup. right
(155 voxels in BA 22, 40, 48)
Calcarine, occipital mid., sup. left
(149 voxels in BA 17)
Paracentral lobule, precuneus left
(56 voxels in BA 2, 4, 5)
Temporal inf. left
(37 voxels in BA 20, 37)

Abbreviations: Brodmann area (BA).
Brain regions and Brodmann areas displaying significant abnormalities of glucose metabolism in ¹⁸F-fluoro-2-deoxy-d-glucose positron emission tomography of patients with anti-leucine rich glioma inactivated 1 protein encephalitis (n = 4, p < 0.005, two sample t-test uncorrected for multiple comparisons and an extent threshold of 30 voxels) in comparison to matched controls (n = 6).

ISSN: 1471-2377