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Table 1 ACE genotype and allele distributions among controls and migraine patients in different studies

From: Angiotensin-converting enzyme gene insertion/deletion polymorphism in migraine patients

  

Genotypes

Alleles

 

N

DD(%)

ID(%)

II(%)

D(%)

I(%)

Controls

      

   Tronvik

403

92 (26.6)

204 (50.6)

107 (22.8)

388 (48.1)

418 (51.9)

   Paterna (ref 8)

201

75 (37.3)

101 (50.3)

25 (12.4)

251 (62.4)

151 (37.6)

   Lea (ref 9)

244

76 (31.1)

122 (50.0)

46 (18.9)

274 (56.1)

214 (43.9)

   Kowa (ref 10)

248

31 (12.5)

114 (46.0)

103 (41.5)

176 (35.5)

320 (64.5)

Migraine

      

   Tronvik

347

78 (22.5)

186 (53.6)

83 (23.9)

342 (49.3)

352 (50.7)

   Paterna

302

146 (48.3)

129 (42.7)

27 (9.0)

421 (69.7)

183 (30.3)

   Lea

250

77 (30.8)

142 (56.8)

31 (12.4)

296 (59.2)

204 (40.8)

   Kowa

176

33 (18.7)

86 (48.9)

57 (32.4)

152 (43.2)

200 (56.8)

MwA subgroup

      

   Tronvik

155

34 (21.9)

87 (56.1)

34 (21.9)

155 (50.0)

155 (50.0)

   Paterna

NA

NA

NA

NA

NA

NA

   Lea

151

48 (31.8)

85 (56.3)

18 (11.9)

181 (59.9)

121 (40.1)

   Kowa

54

14 (25.9)*

26 (48.2)

14 (25.9)

54 (50.0)*

54 (50.0)

MoA subgroup

      

   Tronvik

187

43 (23.0)

96 (51.3)

48 (25.7)

182 (48.7)

192 (51.3)

   Paterna

302

146 (48.3)*

129 (42.7)

27 (9.0)

421 (69.7)

183 (30.3)

   Lea

99

29 (29.3)

57 (57.6)

13 (13.1)

115 (58.1)

83 (41.9)

   Kowa

122

19 (15.6)

60 (49.2)

43 (35.2)

98 (35.2)

146 (59.8)

  1. * Reported significant finding for genotype or allele frequencies