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Table 1 Clinical data

From: Trigeminal isolated sensory neuropathy (TISN) and FOSMN syndrome: despite a dissimilar disease course do they share common pathophysiological mechanisms?

Patient Gender Onset age (years) Onset Duration (years) Clinical course Pain
TISN
1 F 41 Unilateral paroxysmal pain 20 After 15 years, bilateral ongoing pain and mild sensory deficits. YES
2 F 55 Bilateral sensory deficit 16 Stable NO
3 M 57 Unilateral paroxysmal and ongoing pain 13 Slowly developing bilateral sensory deficit and ongoing pain. YES
4 M 63 Unilateral paroxysmal pain 8 Slowly developing bilateral sensory deficit and ongoing pain. YES
5 F 23 Unilateral sensory deficit 8 After 5 years bilateral sensory deficit. NO
6 F 77 Bilateral paroxysmal and ongoing pain 7 Slowly developing bilateral sensory deficit and ongoing pain. YES
7 M 55 Bilateral sensory deficit 6 After 6 years bilateral sensory deficit. NO
8 F 63 Unilateral paroxysmal and ongoing pain 6 After 5 years bilateral sensory deficit. YES
FOSMN
9 M 49 Unilateral ongoing pain 15† Pain and sensory deficits progressively became bilateral. Motor disturbances began after 6 years. Death 15 years after onset of sensory symptoms. YES
10 M 50 Bilateral ongoing pain 13 Sensory deficits progressively developed bilaterally. Motor disturbances began after 4 years. In evolution. YES
11 F 58 Bilateral ongoing pain 8 Sensory deficits progressively developed bilaterally. Motor disturbances began after 2 years. In evolution. YES
12 F 53 Bilateral sensory deficit 8 Motor disturbances began after 6 years. In evolution. NO
13 F 56 Unilateral ongoing pain 6† Pain and sensory deficits progressively became bilateral. Motor disturbances began after 4 years. Death 6 years after onset of sensory symptoms. YES
  1. TISN: Trigeminal isolated sensory neuropathy. FOSMN: Facial onset sensory-motor neuronopathy. †deceased.