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Table 3 Association between SNPs and LAA using the additive, genotype, dominant, and the recessive models

From: Association study of polymorphisms in the ABO gene with ischemic stroke in the Chinese population

SNP Model Allele or geno case control Unadjusted OR (95 % CI) Unadjusted
p-value
Adjusted
OR (95 % CI)
Adjusted p-value
rs579459 Additive C/T 197/721 334/1576 1.29 (1.06-1.57) 0.01 1.27 (1.00-1.61) 0.047
  Dominant CC + CT/TT 174/285 300/655 1.33 (1.06-1.68) 0.015 1.28 (0.96-1.70) 0.09
  Recessive CC/CT + TT 23/436 34/921 1.43 (0.83-2.46) 0.19 1.70 (0.88-3.29) 0.11
rs651007 Additive T/C 193/703 337/1575 1.28 (1.05-1.56) 0.01 1.30 (1.02-1.64) 0.033
  Dominant TT + CT/CC 170/278 304/652 1.31 (1.04-1.66) 0.02 1.29 (0.97-1.71) 0.08
  Recessive TT/CT + CC 23/425 33/923 1.51 (0.88-2.61) 0.13 1.89 (0.97-3.65) 0.06
rs529565 Additive C/T 374/518 709/1201 1.22 (1.04-1.44) 0.01 1.24 (1.02-1.51) 0.03
  Dominant CC + CT/TT 295/151 565/390 1.35 (1.07-1.71) 0.01 1.39 (1.05-1.84) 0.02
  Recessive CC/CT + TT 79/367 144/811 1.21 (0.90-1.64) 0.21 1.24 (0.86-1.79) 0.25
  1. All SNPs were analyzed under additive, genotype, dominant (Dom) and recessive (Rec) models; OR: odds ratio; CI: confidence interval; unadjusted P-value from t-test; adjusted P-value using logistic regression analysis with age, gender, hypertension, diabetes, and dyslipidemia as covariates. Significant P values (p < 0.05) are in bold and p* < 0.017 (Bonferroni multiple correction threshold)