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Table 1 Demographic and clinical information of the study sample

From: The congruency of neuropsychological and F18-FDG brain PET/CT diagnostics of Alzheimer’s Disease (AD) in routine clinical practice: insights from a mixed neurological patient cohort

Groups

AD

non-AD

  

non-AD subgroups

  

p-value

  

PD/PDD

LBD

VD

Unconspicuous

Other

AD vs. non-AD

Participants (N)

33

94

27

16

14

15

22

 

Sex (N: male/female)

18/15

53/41

16/11

13/3

6/8

8/7

10/12

.856

Age (years: M, SD)

76.70, 8.29

74.33, 8.36

73.81, 7.95

77.94, 4.91

77.93, 7.09

77.40, 6.07

67.95, 9.48

.163

Education (years: M, SD)

11.09, 1.91

12.53, 2.98

11.89, 2.72

14.44, 3.35

11.14, 2.18

12.80, 2.93

12.64, 2.95

.011*

MMSE (score: M, SD)

23.76, 4.01

25.30, 3.66

26.00, 2.71

22.13, 4.51

25.57, 3.69

27.06, 1.03

25.43, 3.93

.040*

  1. Note. Classification of AD versus non-AD and non-AD subgroups based on FDG-PET/CT results. AD Alzheimer Disease, LBD Lewy Body Dementia, MMSE Mini Mental Status Examination, PD Parkinson Disease, PDD Parkinson Disease Dementia, VD ascular Dementia, *significant at p-value < .05, two-tailed, based on one-way ANOVA model. The AD group included N = 5 cases with mixed etiology, where AD coincided with cerebrovascular disease (N = 3), hydrocephalus occlusus (N = 1), or where it was classified as atypical AD (N = 1). In the non-AD subgroups, the subgroup “other” involved reports of functional anomalies suspected to be of cancerous or post-operative etiology (n = 7), progressive supranuclear palsy (n = 4), normal pressure hydrocephalus (n = 3), multiple system atrophy type C (n = 3), frontotemporal dementia (n = 2) and unspecified etiology (n = 3)