Biochemical analysis | | LDH | CRP | SAA | KL-6 |
onset | 354 | 0.9 | 68.8 | 1152 |
after treatment | 272 | 0.1 | 7.3 | 469 |
Blood gas analysis | | pH | pCO2 | pO2 | HCO3 |
onset | 7.41 | 36.9 | 68.2 | 22.3 |
Pulmonary function test | | % VC | FEV1.0% | DLCO | |
onset | 79.8 | 70.4 | 49.2 | |
after treatment | 95.9 | 73.3 | 105.3 |
- CRP C-reactive protein, mg/dL, DLCO Diffusing capacity of the lung carbon monoxide, ml/min/mmHg, FEV1.0% Forced expiratory volume percent in one second, %, KL-6 Krebs von den Lungen-6, U/mL, LDH lactate dehydrogenase, U/L, pCO2: mmHg, pO2: mmHg, SAA Serum amyloid A, mg/mL, SP-D surfactant proteins D, ng/mL, %VC Percent vital capacity, %,
- On his admission, routine biochemical analyses revealed increased levels related to IP. Arterial blood gas analysis revealed hypoxemia. Pulmonary function test revealed diffusion impairment. The symptoms and respiratory failure improved, with DLCO increasing after discontinuation and steroid treatment
