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Fig. 1 | BMC Neurology

Fig. 1

From: Is diabetic striatopathy the culprit of seizures in a patient with ketotic hyperglycemia-induced hemichorea–hemiballismus?

Fig. 1

A High T1 signal intensity in the left lentiform nucleus with no significant diffusion restriction or postcontrast enhancement. B, C There are significant atrophic changes of the left hippocampus and left fornix with a subtle increase in signal intensity in FLAIR seen predominantly at the body, loss of interdigitation, and internal architecture bilaterally representing mesial temporal sclerosis. D, E Diffuse diffusion restriction, bright signal intensity on T2/FLAIR with faint postcontrast enhancement involving the left hippocampus and left temporal lobe cortex extending to the left insular and adjacent frontal cortices, likely representing postictal changes

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BMC Neurology

ISSN: 1471-2377

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